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Registro completo
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Biblioteca (s) : |
INIA Treinta y Tres. |
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Fecha : |
19/11/2015 |
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Actualizado : |
08/03/2018 |
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Tipo de producción científica : |
Actividades de Difusión |
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Autor : |
JORNADA ANUAL ARROZ-SOJA, 1983, CIAAB-EEE, TREINTA Y TRES, UY.; CHEBATAROFF, N.; DEAMBROSI, E.; BLANCO, P.H.; ÁVILA, S.; MÉNDEZ, R.; YANG, C.W.; WANG, W.J.; LAVECCHIA, A. |
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Afiliación : |
NICOLÁS CHEBATAROFF; ENRIQUE GERMAN DEAMBROSI CHURRUT, INIA (Instituto Nacional de Investigación Agropecuaria), Uruguay; PEDRO HORACIO BLANCO BARRAL, INIA (Instituto Nacional de Investigación Agropecuaria), Uruguay; MARIFLOR STELLA ÁVILA SILVA, INIA (Instituto Nacional de Investigación Agropecuaria), Uruguay; RAMÓN FELIPE MÉNDEZ LARROSA, INIA (Instituto Nacional de Investigación Agropecuaria), Uruguay; ANDRES PASCUAL LAVECCHIA GONZALEZ, INIA (Instituto Nacional de Investigación Agropecuaria), Uruguay. |
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Título : |
Arroz-soja: Resultados de la experimentación regional en cultivos 1982-83. |
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Fecha de publicación : |
1983 |
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Fuente / Imprenta : |
Treinta y Tres (Uruguay): CIAAB; EEE, 1983 |
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Páginas : |
104 p. |
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Idioma : |
Español |
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Notas : |
Actividad realizada en Agosto de 1983. |
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Contenido : |
ARROZ
Mejoramiento
Épocas de siembra. Ensayos varietales. Ensayos regionales varietales. Respuesta varietal a nitrògeno. Convenio de Cooperación Técnico Agrícola con la República de China. Situación actual y perspectivas del mejoramiento genético de arroz en Uruguay.
Manejo
Ensayos regionales de fertilización. Manejo de nitrógeno. Discusión general de fertilización. Tesis de graduación. Ensayos especiales. Control de maleza.
Fitopatologìa
Camas de infección. Propagaciòn y daño provocado por enfermedades del tallo. Evaluación de fungicidas a campo. Evaluación de fungicidas en laboratorio.
SOJA
Ensayos varietales. Fertilización.
EVALUACIÒN DE DOS SISTEMAS INTENSIVOS DE PRODUCCIÓN EN SUELOS ARROCEROS.
DATOS CLIMÁTICOS |
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Palabras claves : |
SISTEMA ARROZ-SOJA; SISTEMAS INTENSIVOS. |
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Thesagro : |
ARROZ; SOJA. |
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Asunto categoría : |
F01 Cultivo |
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URL : |
http://www.ainfo.inia.uy/digital/bitstream/item/5254/1/Arroz-Soja1982-1983.pdf
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Marc : |
LEADER 01562nam a2200277 a 4500
001 1053942
005 2018-03-08
008 1983 bl uuuu u00u1 u #d
100 1 $aJORNADA ANUAL ARROZ-SOJA, 1983, CIAAB-EEE, TREINTA Y TRES, UY.
245 $aArroz-soja$bResultados de la experimentación regional en cultivos 1982-83.$h[electronic resource]
260 $aTreinta y Tres (Uruguay): CIAAB; EEE$c1983
300 $a104 p.
500 $aActividad realizada en Agosto de 1983.
520 $aARROZ Mejoramiento Épocas de siembra. Ensayos varietales. Ensayos regionales varietales. Respuesta varietal a nitrògeno. Convenio de Cooperación Técnico Agrícola con la República de China. Situación actual y perspectivas del mejoramiento genético de arroz en Uruguay. Manejo Ensayos regionales de fertilización. Manejo de nitrógeno. Discusión general de fertilización. Tesis de graduación. Ensayos especiales. Control de maleza. Fitopatologìa Camas de infección. Propagaciòn y daño provocado por enfermedades del tallo. Evaluación de fungicidas a campo. Evaluación de fungicidas en laboratorio. SOJA Ensayos varietales. Fertilización. EVALUACIÒN DE DOS SISTEMAS INTENSIVOS DE PRODUCCIÓN EN SUELOS ARROCEROS. DATOS CLIMÁTICOS
650 $aARROZ
650 $aSOJA
653 $aSISTEMA ARROZ-SOJA
653 $aSISTEMAS INTENSIVOS
700 1 $aCHEBATAROFF, N.
700 1 $aDEAMBROSI, E.
700 1 $aBLANCO, P.H.
700 1 $aÁVILA, S.
700 1 $aMÉNDEZ, R.
700 1 $aYANG, C.W.
700 1 $aWANG, W.J.
700 1 $aLAVECCHIA, A.
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Registro original : |
INIA Treinta y Tres (TT) |
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 | Acceso al texto completo restringido a Biblioteca INIA La Estanzuela. Por información adicional contacte bib_le@inia.org.uy. |
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Registro completo
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Biblioteca (s) : |
INIA La Estanzuela. |
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Fecha actual : |
02/03/2017 |
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Actualizado : |
07/03/2017 |
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Autor : |
GARCIA, J.P; GIANNITTI, F.; FINNIE, J.W.; MANAVIS, J. .; BEINGESSER, J.; ADAMS ,V.; ROOD, J.I.; UZAL, F.A. |
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Afiliación : |
FEDERICO GIANNITTI, INIA (Instituto Nacional de Investigación Agropecuaria), Uruguay. |
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Título : |
Comparative Neuropathology of Ovine Enterotoxemia Produced by Clostridium perfringens Type D Wild-Type Strain CN1020 and Its Genetically Modified Derivatives. |
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Fecha de publicación : |
2015 |
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Fuente / Imprenta : |
Veterinary Pathology, 2015, v.52.n.6. p.1250-1253, 2015. |
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Idioma : |
Inglés |
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Contenido : |
Abstract
Clostridium perfringens type D causes enterotoxemia in sheep and goats. The disease is mediated by epsilon toxin (ETX), which affects the cerebrovascular endothelium, increasing vascular permeability and leading to cerebral edema. In the present study, we compared the distribution and severity of the cerebrovascular changes induced in lambs by C. perfringens type D strain CN1020, its isogenic etx null mutant, and the ETX-producing complemented mutant. We also applied histochemical and immunohistochemical markers to further characterize the brain lesions induced by ETX. Both ETX-producing strains induced extensive cerebrovascular damage that did not differ significantly between each other in nature, neuroanatomic distribution, or severity. By contrast, lambs inoculated with the etx mutant or sterile, nontoxic culture medium did not develop detectable brain lesions, confirming that the neuropathologic effects observed in these infections are dependent on ETX production. Lambs treated with the wild-type and complemented strains showed perivascular and mural vascular edema, as well as serum albumin extravasation, particularly severe in the cerebral white matter, midbrain, medulla oblongata, and cerebellum. Brains of animals inoculated with the ETX-producing strains showed decreased expression of glial fibrillary acidic protein and increased expression of aquaporin-4 in the end-feet processes of the astrocytes around blood vessels. Early axonal injury was demonstrated with anti?amyloid precursor protein immunohistochemistry. Perivascular accumulation of macrophages/microglia with intracytoplasmic albumin globules was also observed in these animals. This study demonstrates that ETX is responsible for the major cerebrovascular changes in C. perfringens type D?induced disease. MenosAbstract
Clostridium perfringens type D causes enterotoxemia in sheep and goats. The disease is mediated by epsilon toxin (ETX), which affects the cerebrovascular endothelium, increasing vascular permeability and leading to cerebral edema. In the present study, we compared the distribution and severity of the cerebrovascular changes induced in lambs by C. perfringens type D strain CN1020, its isogenic etx null mutant, and the ETX-producing complemented mutant. We also applied histochemical and immunohistochemical markers to further characterize the brain lesions induced by ETX. Both ETX-producing strains induced extensive cerebrovascular damage that did not differ significantly between each other in nature, neuroanatomic distribution, or severity. By contrast, lambs inoculated with the etx mutant or sterile, nontoxic culture medium did not develop detectable brain lesions, confirming that the neuropathologic effects observed in these infections are dependent on ETX production. Lambs treated with the wild-type and complemented strains showed perivascular and mural vascular edema, as well as serum albumin extravasation, particularly severe in the cerebral white matter, midbrain, medulla oblongata, and cerebellum. Brains of animals inoculated with the ETX-producing strains showed decreased expression of glial fibrillary acidic protein and increased expression of aquaporin-4 in the end-feet processes of the astrocytes around blood vessels. Early axonal injury was demonstrated wi... Presentar Todo |
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Thesagro : |
ENFERMEDADES DE LOS ANIMALES. |
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Asunto categoría : |
-- |
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Marc : |
LEADER 02512naa a2200217 a 4500
001 1056750
005 2017-03-07
008 2015 bl uuuu u00u1 u #d
100 1 $aGARCIA, J.P
245 $aComparative Neuropathology of Ovine Enterotoxemia Produced by Clostridium perfringens Type D Wild-Type Strain CN1020 and Its Genetically Modified Derivatives.$h[electronic resource]
260 $c2015
520 $aAbstract Clostridium perfringens type D causes enterotoxemia in sheep and goats. The disease is mediated by epsilon toxin (ETX), which affects the cerebrovascular endothelium, increasing vascular permeability and leading to cerebral edema. In the present study, we compared the distribution and severity of the cerebrovascular changes induced in lambs by C. perfringens type D strain CN1020, its isogenic etx null mutant, and the ETX-producing complemented mutant. We also applied histochemical and immunohistochemical markers to further characterize the brain lesions induced by ETX. Both ETX-producing strains induced extensive cerebrovascular damage that did not differ significantly between each other in nature, neuroanatomic distribution, or severity. By contrast, lambs inoculated with the etx mutant or sterile, nontoxic culture medium did not develop detectable brain lesions, confirming that the neuropathologic effects observed in these infections are dependent on ETX production. Lambs treated with the wild-type and complemented strains showed perivascular and mural vascular edema, as well as serum albumin extravasation, particularly severe in the cerebral white matter, midbrain, medulla oblongata, and cerebellum. Brains of animals inoculated with the ETX-producing strains showed decreased expression of glial fibrillary acidic protein and increased expression of aquaporin-4 in the end-feet processes of the astrocytes around blood vessels. Early axonal injury was demonstrated with anti?amyloid precursor protein immunohistochemistry. Perivascular accumulation of macrophages/microglia with intracytoplasmic albumin globules was also observed in these animals. This study demonstrates that ETX is responsible for the major cerebrovascular changes in C. perfringens type D?induced disease.
650 $aENFERMEDADES DE LOS ANIMALES
700 1 $aGIANNITTI, F.
700 1 $aFINNIE, J.W.
700 1 $aMANAVIS, J. .
700 1 $aBEINGESSER, J.
700 1 $aADAMS ,V.
700 1 $aROOD, J.I.
700 1 $aUZAL, F.A.
773 $tVeterinary Pathology, 2015$gv.52.n.6. p.1250-1253, 2015.
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